Extraboline

[hommerich]

Ik heb net 20 vials extraboline aangekocht, het ziet er alleszins echt uit, ik heb meerdere malen gecheckt op enkele websites ( ook bodypage ).

Laten we ervan uitgaan dat ie echt is. Maar ik heb meerdere malen gelezen dat Genepharm de nandrolone serieus onderdoseerd, is dit waar.

Heeft iemand daar soortgelijke verhalen van gehoord in hoeverre is dit betrouwbaar.

Gebruiken doe ik ze toch

[ron2108]

Heb vorig jaar een kuurtje met alleen extraboline gedaan,maar dit leverde noppes op ,alleen een deca dick (maar dit kon ook tussen mijn oren hebben gezeten..
Gr r

[Adonis]

ik denk niet dat je door alleen te denken een deca-dick op loopt.... Dus misschien gewoon ondergedoseerd

[bruce]

van nandrolone only (deca only) krijg je in 90% van de gevallen toch een d-dick. hoe meer erin zit, hoe erger de dick. ofwel: alstie ondergedoseerd is heb je minder last. maar ja ook minder gains. alleen nandrolone is voor mij sowieso geen optie. altijd test. erbij.

[hommerich]

Ik zal je ervan op de hoogte houden bruce want ik begin bijna met mijn kuur 2ml susta, 3ml drive, 4ml extra, gespreid over susta+extra en drive+extra in de week. en dit 8 weken lang, ditmaal zal ik zeker nakuren hcg+nolva/clomid.

[ron2108]

drive +extra

[bruce]

extra is extraboline.
graag, hom. ik ben benieuwd. flinke kuur dus.

[hommerich]

zeg Bruce over nakuren worden er dikwijls tegenovergestelde dingen gezegd vooral ivm met hcg, de ene zegt dat je het moet nemen vanaf week 5 500/1000 ui om de 3,4 dagen (gelezen op bodypage) en hier op het forum neemt men hcg op het laatste, terwijl ik artikels lees dat je dit niet mag doen.

Graag een goed sluitend antwoord aub.

Bodynet rules

[bruce]

hm. als je alle posts had gelezen was mijn mening er missch. uit gekomen.

nakuren niet met hcg. hcg is alleen nodig om (als jet zelf dus nodig vindt) je ballen weer groter te krijgen.
als je hcg tijdens je kuur gebruikt, dan alleen als je gekrompen ballen hebt. anders heeft het weinig zin.

dus het kan na je kuur, maar niet zonder je nakuur. die moet je evengoed doen.

[hommerich]

ik heb al vele posts gelezen maar ik geraak er niet aan uit vooral dat artikel

zie forum kuren en resultaten; aan mr C; post van Black cool ...

[Jack]

Citaat[/b] (bruce @ Jan. 20 2004,20:10)]hm. als je alle posts had gelezen was mijn mening er missch. uit gekomen.

nakuren niet met hcg. hcg is alleen nodig om (als jet zelf dus nodig vindt) je ballen weer groter te krijgen.
als je hcg tijdens je kuur gebruikt, dan alleen als je gekrompen ballen hebt. anders heeft het weinig zin.

dus het kan na je kuur, maar niet zonder je nakuur. die moet je evengoed doen.

uehhh bruce,

hcg is niet nodig? bij kleine kuurtjes niet nee, maar bij zwaardere kuren is het zeker wel aan te raden.
lees dit stuk maar eens ... Understanding Post Cycle 'T' Recovery

Onderin dat artikel staat een goede nakuur voor na een zware kuur die je axis goed heeft onderdrukt.

[Myobolix]

Understanding Post Cycle “T” Recovery
by William Llewellyn


O.K. You have been on an awesome 4-month cycle of Sustanon and Dianabol. You’ve gained a massive 20 lbs, and are extremely pleased with your results. You can’t stop looking in the mirror. But there is a problem now starting to eat away at you. You are going to run out of steroids very soon (you know you need a break anyway), and your testicles are the size of raisins. Your body is producing less testosterone than a 9-year-old girl, and you are scrambling to figure out what to do to avoid a nasty post-cycle crash that could potentially strip away some of your hard-earned muscle. The opinions on how to restore endogenous testosterone production post-cycle seem to be different everywhere you look. What option is best? Without an understanding of exactly what is going on in your body, and why certain compounds help to correct the situation, choosing the right post-cycle program can be quite confusing. In this article I would therefore like to discuss the role of anti-estrogens and HCG during this delicate window of time, while detailing an effective strategy for their use.



The Axis


The Hypothalamic-Pituitary-Testicular Axis, or HPTA for short, is the thermostat for your body’s natural production of testosterone. Too much testosterone and the furnace will shut off. Not enough, and the heat is turned up, to put it very simply. For the purposes of our discussion here we can look at this regulating process as having three levels. At the top is the hypothalamic region of the brain, which releases the hormone GnRH (Gonadotropin-Releasing Hormone) when it senses a need for more testosterone. GnRH sends a signal to the second level of the axis, the pituitary, which releases Luteinizing Hormone in response. LH for short, this hormone stimulates the testes (level three) to secrete testosterone. The same sex steroids (testosterone, estrogen) that are produced serve to counter-balance things, by providing negative feedback signals (primarily to the hypothalamus and pituitary) to lower the secretion of testosterone when too much of this hormone is sensed. Synthetic steroids, of course, suppress testosterone the same way. This quick background of the testosterone-regulating axis is necessary to furthering our discussion, as we need to first look at the underlying mechanisms involved before we can understand why natural recovery of the HPTA post-cycle is a slow process. Only then can we implement an ancillary drug program to effectively deal with it.



Testicular Desensitization


Although steroids suppress testosterone production primarily by lowering the level of gonadotropic hormones discussed above, the big roadblock to a restored HPTA after we come off the drugs is surprisingly not the level of LH itself. This problem is made clearly evident in a study published in Acta Endocrinologica back in 1975(1). Here blood parameters, including testosterone and LH levels, were monitored in male subjects whom were given testosterone enanthate injections of 250mg weekly for 21 weeks. Subjects remained under investigation for an additional 18 weeks after the drug was discontinued. At the start of the study, LH levels became suppressed in direct relation to the rise in testosterone, which is to be expected. Things looked very different, however, once the steroids had been withdrawn (see Figure I). LH levels went on the rise quickly (by the 3rd week), while testosterone barely budged for quite some time. In fact, on average it was more than 10 weeks before any noticeable movement started. This lack of correlation makes clear that the problem in getting androgen levels restored is not the level of LH, but in fact testicular atrophy and desensitization to this hormone. After a period of inactivation the testes have apparently lost mass (atrophied), making them unable to perform the workload required by heightened levels of LH.


Post-Cycle LH Levels


Post Cycle Testosterone Levels

Figure I. LH and Testosterone measurements starting 1 week after the last injection of 250mg of testosterone enanthate (pretreated measures were 5 mU/ml and 4.5 ng/ml respectively). Note that between weeks 1 and 5, as testosterone levels are declining due to the cessation of exogenous androgen administration, LH levels are already rebounding. From weeks 5 to 10 testosterone levels are at or very near baseline, to spite the substantial LH levels by this point. No significant increase in testosterone is noted until after the 10-week mark.



The Role of Anti-estrogens


It is important to understand that anti-estrogens alone do not do much to restore endogenous testosterone release after a cycle. Normally they only foster LH by blocking the negative feedback of estrogens, and we now see that LH rebounds quickly without help anyway. Plus, post cycle there is not an elevated level of estrogen for anti-estrogens to block, as testosterone (now suppressed) is a major substrate used for the synthesis of estrogens in men. Serum estrogen levels will actually be lower here as a result, not higher. Any estrogen rebound that occurs post-cycle likewise happens concurrently with a rebound in testosterone levels, not prior to it (note there is an imbalance in the ratio post cycle, but this is another topic altogether). We are seeing no mechanism in which anti-estrogenic drugs can really help here. We can see why this fact would not be difficult to overlook, however. The medical literature is filled with references showing anti-estrogenic drugs like Clomid and Nolvadex to increase LH and testosterone levels, and in normal situations these drugs do indeed increase endogenous androgen production by blocking the negative feedback of estrogens. Combine this with the fact that just as many studies can be found to show that steroid use lowers LH levels when suppressing testosterone, and we can see how easy it would be to jump to the conclusion that post-cycle we need to focus on restoring LH. We would miss the true problem of testicular desensitization unless we were really looking into the actual recovery rates of the hormones involved. When we do, we immediately see little value in using anti-estrogenic drugs.



HCG


So we now see, contrary to the dominating opinion of the times, that anti-estrogens alone will do little to raise testosterone levels in the early weeks of the post-cycle window. This leaves us to focus on a very different level of the HPTA in order to hasten recovery: the testes. For this we will need the injectable drug HCG. If you are not familiar with it, HCG, or Human Chorionic Gonadotropin, is a prescription fertility agent that mimics the bodies own natural LH. Although the testes are equally desensitized to this drug as LH (they both work through the same mechanism), we are administering it as a measured drug and are therefore not constrained by the limits of our own LH production. We similarly can use HCG to provide a bolus dose of LH (of our choosing), which works only to augment the recovering LH levels we already have in the body. In essence we are looking to shock them with an overwhelmingly high level of LH activity, coming from both endogenous and exogenous sources. We want it to reach a level far above what our body, even when supported by anti-estrogens, could possibly do on its own. The result can be a rapid restoration of original testicular mass and functioning, which would allow normal levels of testosterone to be output much sooner than without such an ancillary program. What we are looking at now is HCG actually being the pivotal post-cycle drug, while anti-estrogens are relegated to a supportive role at best.



Finalizing the Program


An ideal post-cycle recovery program will focus on two things really. The first is hitting the testes hard with HCG. It is important, however, not to overuse this drug. Taken for too long, or at too high a dosage, the LH receptor will actually become desensitized to LH(2) , which may further exacerbate our post-cycle problem instead of helping it (this is why I am not in favor of regular HCG use on-cycle). My experience with HCG has led me to feel comfortable using it for a course of three weeks, at a dosage of maybe 5000-7500IU weekly. Often the last week I limit the dose to 2,500IU, unless the cycle has been particularly long or potent. This is timed so at least half of the total administered drug dosage will be given when there is still exogenous steroid in the body. On our graph above this would be at about the 3-week mark after the last injection of testosterone. This will give the testes some time to get back into shape before the baseline is actually hit with T levels. Secondly, Anti-estrogens are used to play a supportive role at the same time, so 20mg of Nolvadex or 50-100mg of Clomid would typically be added (my last article for Mind and Muscle discusses the comparative differences with these two agents). This is to combat the suppressive effects of estrogen as testosterone levels start to go back up, as well as potential side effects (HCG has been shown to increase testicular aromatase activity as well (3)). Although in the first couple of weeks the anti-estrogen does little, it may indeed be helpful when testosterone levels actually start to get back up near normal. To further stimulate the HPTA, and support continuingly high LH levels, the anti-estrogen remains to be used for 2 to 3 weeks after the HCG therapy has been stopped. A sample program, as it would be instituted in our sample post-cycle window, is provided below.



Sample Post-cycle Plan:


Week
Amount

Week 3:
5000IU HCG total + 20mg Nolvadex daily

Week 4:
5000IU HCG total + 20mg Nolvadex daily

Week 5:
2500IU HCG total + 20mg Nolvadex daily

Week 6:
20mg Nolvadex daily

Week 7:
20mg Nolvadex daily

Week 8:
20mg Nolvadex daily





In Closing


I hope this article provided a well-needed new look at the mechanisms involved in post-cycle testosterone recovery. Indeed I believe it should debunk a commonly held belief these days, as we seen now that those advocating the sole use of Clomid post cycle are sorely missing the mark. The problem goes much deeper than just getting LH levels back. In fact, we see that LH doesn’t even need much help kicking back into gear, and a drug like Clomid will do very little to help this anyway in the absence of significant estrogen levels anyway. HCG is a drug with undeniable usefulness during the post-cycle window, and many bodybuilders have been much too quick to abandon it. It is truly fundamental to an effective recovery program, and would not consider any dose or combination of anti-estrogens or aromatase inhibitors capable of doing the job without it.

[Myobolix]

Citaat[/b] ]Secondly, Anti-estrogens are used to play a supportive role at the same time, so 20mg of Nolvadex or 50-100mg of Clomid would typically be added

Goed artikel!
Alleen dit gequote gedeelte heb ik wel moeite mee. Het invoegen van Clomid of nolvadex hou je best voor NA je hcg toediening (gedurende 2-tal weken) en dit omdat het daarvoor gemaakt is, namelijk het terug omhoogbrengen van de endogene LH waardes na de toediening van exogeen LH dwz HCG.

We gaan onze eigen LH produktie stopzetten door HCG en terug activeren door erna Clomid en Nolvadex te gebruiken. Tijdens HCG is zinloos.

Citaat[/b] ]This is to combat the suppressive effects of estrogen as testosterone levels start to go back up, as well as potential side effects (HCG has been shown to increase testicular aromatase activity as well

Welnu, hiervoor kan je tijdens de HCG beter iets anders nemen, een aromatase remmer als Arimidex. Je kan dit zelfs best beginnen, 4-tal dagen voor begin kuur en verder nemen gedurende hele kuur, na-kuur tot voor de dag dat je begint met clomid, nolvadex.

Gewoon een kwestie van logisch nadenken, meer niet...

[Myobolix]

Dit artikel, met de commentaar over clomid en nolva zou best een "sticky" worden worden zodat iedereen ten allen tijden hiernaar kan terugwijzen, niet?

Basis kennis!

[bruce]

Citaat[/b] (Jack @ Jan. 21 2004,09:21)]

Citaat[/b] (bruce @ Jan. 20 2004,20:10)]hm. als je alle posts had gelezen was mijn mening er missch. uit gekomen.

nakuren niet met hcg. hcg is alleen nodig om (als jet zelf dus nodig vindt) je ballen weer groter te krijgen.
als je hcg tijdens je kuur gebruikt, dan alleen als je gekrompen ballen hebt. anders heeft het weinig zin.

dus het kan na je kuur, maar niet zonder je nakuur. die moet je evengoed doen.

uehhh bruce,

hcg is niet nodig? bij kleine kuurtjes niet nee, maar bij zwaardere kuren is het zeker wel aan te raden.
lees dit stuk maar eens ... Understanding Post Cycle 'T' Recovery

Onderin dat artikel staat een goede nakuur voor na een zware kuur die je axis goed heeft onderdrukt.

dan heb ik zeker nog nooit zwaar gekuurd. wat is zwaar. ik blijf erbij dat het voor te kleine borrelnootjes is. (wat je uiteraard in een zware kuur erger hebt dan een licht kuurtje)