@ Healthenfitness
Geen zaken door elkaar gooien kerel. 5-HTP is een product dat afkomt van L-Tryptofaan, maar zeker niet hetzelfde. Lees mijn artikel er even over, daar leg ik het keurig uit.
@ Erik77
Ik betwijfel of dat problemen gaat geven. Ik heb hieronder een studie naar 1-5-HTP (en ik heb waarlijk geen idee of dat een andere vorm is van 5-HTP, of dat het gewoon een uitgebreidere schrijfwijze is), maar die nuanceert het verhaal ie****************:
Geen zaken door elkaar gooien kerel. 5-HTP is een product dat afkomt van L-Tryptofaan, maar zeker niet hetzelfde. Lees mijn artikel er even over, daar leg ik het keurig uit.
@ Erik77
Ik betwijfel of dat problemen gaat geven. Ik heb hieronder een studie naar 1-5-HTP (en ik heb waarlijk geen idee of dat een andere vorm is van 5-HTP, of dat het gewoon een uitgebreidere schrijfwijze is), maar die nuanceert het verhaal ie****************:
Effect of 1-5 hydroxytryptophan infusion on growth hormone and prolactin secretion in man.
Lancranjan I, Wirz-Justice A, Pühringer W, Del Pozo E.
Abstract
The effect of a new soluble ester of 1-5 hydroxytryptophan (1-5 HTP, Ro 3-5940, 200 mg infusion) on prolactin (PRL) and growth hormone (GH) release was tested in 11 young, healthy subjects (6 men, 5 women). To minimize side-effects, peripheral decarboxylase inhibition was achieved with benserazide (Ro 4-4602.) PRL increased significantly (P less than 0.01) after benserazide alone in all subjects. A further significant increase (P less than 0.01) of PRL plasma levels occurred only in women up to 90 min after the infusion of 1-5 HTP was discontinued. Benserazide administration had no effect of basal GH levels, but a significant increase of GH release (P less than 0.01) was noticed 30-120 min after the end of 1-5 HTP infusion in both men and women. The mean peak value of GH plasma levels after 1-5 HTP administration was 32.0 +/- 8.8 ng/ml. It was postulated that benserazide penetrated at the level of the pituitary, decreasing the synthesis of dopamine and consequently reducing its known inhibitory effect on PRL release. The PRL increase (statistically significant only in women), as well as the release of GH after 1-5 HTP infusion, was considered as further evidence for stimulatory serotoninergic control of both PRL and GH secretion.
PMID: 302840 [PubMed - indexed for MEDLINE]
Lancranjan I, Wirz-Justice A, Pühringer W, Del Pozo E.
Abstract
The effect of a new soluble ester of 1-5 hydroxytryptophan (1-5 HTP, Ro 3-5940, 200 mg infusion) on prolactin (PRL) and growth hormone (GH) release was tested in 11 young, healthy subjects (6 men, 5 women). To minimize side-effects, peripheral decarboxylase inhibition was achieved with benserazide (Ro 4-4602.) PRL increased significantly (P less than 0.01) after benserazide alone in all subjects. A further significant increase (P less than 0.01) of PRL plasma levels occurred only in women up to 90 min after the infusion of 1-5 HTP was discontinued. Benserazide administration had no effect of basal GH levels, but a significant increase of GH release (P less than 0.01) was noticed 30-120 min after the end of 1-5 HTP infusion in both men and women. The mean peak value of GH plasma levels after 1-5 HTP administration was 32.0 +/- 8.8 ng/ml. It was postulated that benserazide penetrated at the level of the pituitary, decreasing the synthesis of dopamine and consequently reducing its known inhibitory effect on PRL release. The PRL increase (statistically significant only in women), as well as the release of GH after 1-5 HTP infusion, was considered as further evidence for stimulatory serotoninergic control of both PRL and GH secretion.
PMID: 302840 [PubMed - indexed for MEDLINE]
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